DEVELOPMENT AND FUNCTION OF THE BLOOD VESSELS: The blood vessels make up the body’s cardiovascular system. The systemic circulation carries oxygenated blood from the left ventricle through the arteries to the various tissues of the organism. In the capillaries, the smallest of blood vessels, oxygen and other nutrients are exchanged for cellular waste and carbon dioxide. The veins take de-oxygenated blood back to the heart and deliver it through the right heart chambers and the pulmonary arteries to the lungs. The pulmonary circulation returns oxygenated blood from the lungs to the left atrium, which empties into the left ventricle, completing the cycle of blood circulation. The blood vessel wall is endowed with connective tissue, smooth muscle, and striated muscles. Equal to the intestinal muscles that move the “food morsel” along the intestinal canal through peristaltic motion, the smooth muscles of the arteries and veins facilitate the flow of the “blood morsel”. The inner lining of the arteries and veins, the so-called intima, originates from the new mesoderm and is therefore controlled from the cerebral medulla.
BIOLOGICAL CONFLICT: The biological conflict linked to the arteries is a light self-devaluation conflict experienced in the area of a particular artery. The specific self-devaluation conflicts are the same as for the bones and joints.
CONFLICT-ACTIVE PHASE: localized necrosis (cell loss) of the artery proportional to the degree and duration of conflict activity. While the intima necrotizes, the smooth muscles of the artery become thicker in order to prevent a perforation of the arterial wall. However, if an intense conflict persists for a long period of time, the blood vessel wall becomes weak causing a localized bulge or aneurysm, for instance, in one of the external carotid arteries (compare with carotid artery aneurysm related to the internal carotid artery). A cerebral aneurysm in other brain arteries than the carotid arteries is extremely rare. The most common location of arterial aneurysms is the abdominal aorta, specifically the segment of the abdominal aorta below the kidneys. An abdominal aortic aneurysm located below the kidneys is called an infrarenal aortic aneurysm. Small aneurysms may go completely unnoticed. However, as the aneurysm becomes larger there is a greater risk of rupture. Normally, the smooth muscle fibers embedded in the striated muscles of the arterial wall stabilize the blood vessel. Hence, an aneurysm rupture only occurs because of a vigorous move, lifting something heavy, or pressing too hard during a bowel movement. Hemorrhaging into the abdomen is a medical emergency. When a cerebral aneurysm bursts, this causes bleeding in the brain (compare with bleeding due to a ruptured brain cyst). A brain hemorrhage, however, is not related to a stroke, as claimed by conventional medicine.
HEALING PHASE: During the first part of the healing phase (PCL-A) the necrotized area in the affected artery is replenished through cell proliferation with localized swelling. Bacteria, if available, assist the healing process, potentially accompanied by an inflammation (arteritis).
The blood vessel is repaired with the help of calcium and cholesterol. With continuous conflict relapses, plaques accumulate at the site leading to arteriosclerosis. Arteriosclerosis in the penile arteries, linked to a sexual self-devaluation conflict, restricts the rush of blood into the penis required to get and maintain an erection; the narrowed blood vessels compromise the function of the erectile system causing erectile dysfunction (see also erectile dysfunction related to the corpora cavernosa). In the major arteries (coronary arteries, ascending aorta, internal carotid arteries, and inner sections of the subclavian arteries) the arteriosclerotic plaques certainly compromise the blood flow but do not cause a heart attack or a stroke, as claimed.
In the legs, the swelling and buildup of plaques narrow the lumen of the artery leading to pain and difficulties walking. Medically this is referred to as peripheral artery disease or “intermittent claudication”. For a person unfamiliar with GNM, the pain usually triggers new self-devaluation conflicts (“My legs are useless”!) resulting in a chronic condition. If the striated muscles of the leg arteries are involved due to a leg-related motor conflict (not being able to run away, not being able to walk because of pain), leg cramps, typically in the calf muscle, occur throughout the Epileptoid Crisis. With ongoing conflict relapses the constant spasms constrict the blood vessel. The narrowing of the affected artery is often diagnosed as a “peripheral artery disease”, even though there are no “diseased” changes of the blood vessel itself.
BIOLOGICAL CONFLICT: Like the arteries, the veins are also linked to a self-devaluation conflict. The specific self-devaluation conflicts are the same as for the bones and joints.
CONFLICT-ACTIVE PHASE: localized necrosis (cell loss) proportional to the degree and duration of conflict activity. While the intima necrotizes, the smooth muscles of the vein become thicker in order to prevent a perforation.
HEALING PHASE: During the first part of the healing phase (PCL-A) the necrotized area in the affected vein is replenished through cell proliferation. With an inflammation (phlebitis) the area around the vein is red, warm, and tender. Bacteria assist the healing process, provided they are available.
The accumulation of fluid in the healing area creates a peripheral edema, for example, in the ankles, feet, and legs (see also peripheral edema related to the myocardium or to the leg bones; compare with lymphedema).
In conventional medicine, pain and swelling in the leg are often misdiagnosed as “deep vein thrombosis” or “thrombophlebitis”, based on the wrong assumption that the swelling and inflammation of the vein is caused by a thrombus.
Varicose veins are a hanging healing in the leg veins caused by continuous conflict relapses. The leg valves that prevent blood from flowing backward are also affected. With recurrent repair processes the valves become scarred (PCL-B) and porous with the result that the veins thicken.
Varicoceles are varicose veins that develop in the scrotum. In this case, the self-devaluation conflict is associated with the testicles. This would explain why varicoceles typically develop in puberty.