DEVELOPMENT AND FUNCTION OF THE SKELETAL MUSCLES: The musculoskeletal system provides form to the body and allows the body to move and maintain its posture. The muscles are connected to the bones and joints through tendons and ligaments and are endowed with connective tissue, nerve tissue, and blood vessels. The skeletal muscles are composed of bundles of fibers that are organized in a striped pattern; this is why they are called striated muscles. Skeletal muscles vary considerably in shape and size. They range from extremely tiny strands such as the stapedius muscle of the middle ear to large masses like the muscle of the thigh. The skeletal muscles originate from the new mesoderm and are controlled from the cerebral medulla and the motor cortex.
The biological conflicts linked to the skeletal muscles is a moderate self-devaluation conflict. The specific self-devaluation conflicts are the same as for the bones and joints.
The conflict related to the movement of the muscles is a motor conflict of “not being able to move” or “feeling stuck”. The conflict can be associated with the entire body (generalized motor conflict) or with a single muscle or muscle group (localized motor conflict).
Leg muscles: not being able to escape, flee, or run away (literally or figuratively, e.g., from a workplace or a relationship), not being able to leap aside, not being able to follow, feeling rooted to the spot (petrified), feeling trapped (literally or figuratively), not being able to keep up, not being able to climb up (e.g., not being promoted), not being able to kick somebody away (extensor muscle), a fear of not being able to walk (wheelchair image).
Motor conflicts can also be experienced with or on behalf of someone else, particularly, when “feeling stuck” concerns a loved one. The belief that conditions such as ALS or MS are hereditary makes a family member more susceptible to conflicts of the same kind (see GNM Article “Understanding Genetic Diseases”).
A fetus might endure the conflict of “not being able to escape” when the mother is in danger or because of threatening noises in the immediate environment (jackhammers, chainsaws, lawn mowers, grass trimmers), loud kitchen equipment such as blenders held close to the womb, or screaming and yelling (fights between parents, mother yelling at her children). In this case, the baby is born with (partial) paralysis of the legs and motor disabilities, if the conflict is not resolved. The "loud noises" of ultrasound examinations can be highly traumatic for the unborn (see Down syndrome). A “feeling stuck”-conflict could be activated during a difficult delivery or the way the baby is handled immediately after birth.
Animals suffer motor conflicts as well, for example, during a fight with another animal, when they are “stuck” in a kennel, tied to a chain, locked in a car, trapped in a cage, or held down by the vet during an examination or vaccination (see conflicts triggered through the practice of animal testing).
CONFLICT-ACTIVE PHASE: cell loss (necrosis) of muscle tissue (controlled from the cerebral medulla) and, at the same time, muscle weakness or muscle paralysis (controlled from the motor cortex). With the impact of the conflict in the motor cortex, less nerve impulses are transmitted to the corresponding muscle causing a loss of muscle function (compare with sensory paralysis related to the epidermis and the periosteum). The biological purpose of the paralysis originates in the fake-death reflex (prey animals “play dead” when they face a predator or danger). The muscle weakness might be noticed as clumsiness or heaviness, when the legs are affected.
Prolonged conflict activity leads to muscle atrophy (muscle wasting) without paralysis if the conflict is experienced solely as a self-devaluation conflict. The pelvic floor muscles become weak because of a difficult pregnancy, sexual humiliation, chronic constipation, or urinary incontinence making the person feel “worthless” there.
With a motor conflict muscle atrophy and muscle paralysis occur together, particularly when the distress of not being able to move an arm or leg (or both) causes a self-devaluation conflict.
Muscle weakness and muscle paralysis were formerly diagnosed as paralytic poliomyelitis, or “polio”, purportedly a “viral infection” that mainly affects children (the scientific evidence of the existence of a “polio virus” has never been provided!). Today, at least in the Western World where polio is supposed to be eradicated by vaccination, the same symptoms are called ALS (Amyotrophic Lateral Sclerosis, also known as Lou Gehrig’s disease or motor neuron disease), Multiple Sclerosis, or Guillain-Barré syndrome (see also renaming of smallpox to pustular eczema after the performance of mass vaccination programs). “Movement disorders” as presented in Parkinson’s and Huntington’s disease are considered inherited “neurodegenerative diseases” (see GNM Article “Understanding Genetic Diseases”).
Multiple Sclerosis (MS)
Muscle weakness and a loss of sensitivity in the feet, legs or arms (see sensory paralysis related to the epidermis and the periosteum) is considered as one of the first symptoms of multiple sclerosis.
Without the knowledge of GNM, an MS diagnosis causes great panic. The same holds true when a person is diagnosed with ALS. The fear of not being able to walk and ending up in a wheelchair (“feeling stuck”) is so overwhelming that the motor conflict which had given rise to the first symptoms often becomes irrelevant. With the progression of the paralysis the muscle atrophy also advances leading to clumsiness, difficulties walking, and frequent falls (see also “falling conflict” and vertigo). This activates additional motor and self-devaluation conflicts with the result that the mobility becomes more and more impaired and the prognosis becomes a self-fulfilling prophecy. The belief that MS, ALS, or Parkinson’s are “hereditary diseases” makes a person whose parent has the condition more vulnerable to suffer a motor conflict (conflicts can also be experienced with someone). The subsequent symptoms lead quickly to the same diagnosis (see GNM Article “Understanding Genetic Diseases”).
In conventional medicine, it is assumed that multiple sclerosis is caused by a “degradation of the myelin sheath” concluded from MRI brain images (the myelin sheath is an insulating layer that envelops nerves, including nerves in the brain and spinal cord). The “myelin destruction” is thought to be an “autoimmune response” where the immune system “mistakenly” destroys the myelin sheath covering the motor neurons in the brain. Like the immune system theory, the concept of “autoimmune disorders” that supposedly damage healthy body tissue is an academic construct that has no scientific basis. The claim that MS is the result of a “destruction” of the myelin sheath is therefore highly questionable.
Vision impairments, which are quite common in people with MS, arise when a brain edema (in PCL-A) or a large glia buildup (in PCL-B) compresses the optic nerve that runs from the retina of the eye through the cerebral medulla to the visual cortex. Optic neuritis, an inflammation of the optic nerve, is therefore often associated with multiple sclerosis. Other vision problems (see retina) are brought on by the fears evoked by the “disease” rather than by the “disease spreading to other organs”, as claimed.
The facial muscles are supplied by the facial nerve (seventh cranial nerve) that also innervates the front two-thirds of the tongue, the upper eyelid muscle, the tear ducts, and the stapedius muscle of the ear. Symptoms of Bell’s palsy therefore include tongue weakness affecting speech and swallowing (tingling or numbness of the tongue and a loss of taste originate from the sensory branch of the facial nerve), incomplete lid closure, excessive tearing, and a heightened sensitivity to sound (hyperacusis). The motor branch of the trigeminus nerve (fifth cranial nerve), which provides motor control to the jaw muscles, affects motor functions such as biting and chewing.
HEALING PHASE: During the healing phase, the atrophied muscle is reconstructed through cell proliferation with swelling due to the edema (fluid accumulation). Concurrent water retention (the SYNDROME) increases the swelling considerably. In conventional medicine, a large swelling is often diagnosed as a muscle sarcoma (myosarcoma) or “soft tissue sarcoma” (see also connective tissue sarcoma).
Muscle hypertrophy, an enlargement of the muscle, is the result of a continuous healing process (hanging healing).
The swelling makes the muscle stiff and tense with pain ranging from mild to severe, depending on the intensity of the conflict-active phase (a “cold” muscle pain points to an involvement of the periosteum; a “hot” muscle pain indicates that the muscle itself is healing). A sore or stiff neck, for instance, reveals an intellectual self-devaluation conflict with difficulties turning the head to one side (see also cervical spine). Which side is affected is determined by a person’s handedness and whether the conflict is mother/child or partner-related. Fibromyalgia is the medical term for widespread muscle pain. In GNM terms, fibromyalgia indicates a long-lasting healing of a generalized self-devaluation conflict affecting the whole person. In conventional medicine, overall muscle pain is also considered a symptom of “chronic fatigue syndrome” (myalgic encephalomyelitis). The persistent tiredness is believed to be caused by an infection with the “Epstein Barr virus” that has also been made responsible for mononucleosis presenting as swollen lymph nodes in the neck. Based on the Second Biological Law, “chronic fatigue” is a symptom that occurs in any prolonged healing phase (vagotonia).
The brain edema that develops in the motor cortex during the first part of the healing phase stretches the synapses between the neurons, which delays the transfer of nerve impulses to the affected muscle(s) even more (see conflict-active phase). As a result, in PCL-A the paralysis remains and the muscle weakness increases! For the uninformed, the further loss of muscle function usually leads to additional motor conflicts and a worsening of the condition. If the conflict-active phase was moderate, the muscle weakness might only be noticed in the healing phase.
During the Epileptoid Crisis, a sympathicotonic surge (visible on an EEG as an electrical discharge) expels the edema in the motor cortex. The sudden reconnection of the nerve cells causes rhythmic convulsions, muscle spasms, muscle cramps, or muscle twitching. The exaggerated muscular movements are a positive sign that the muscle function is striving to get back to normal.
An intense Epileptoid Crisis manifests as an epileptic seizure with tonic-clonic convulsions and rapid muscle contractions. A localized or focal seizure with spasms or jerking of a single muscle or muscle groups is confined to the conflict-related muscles, for example in the leg(s) or arm(s). In a grand mal seizure, the convulsions involve the muscles of the whole body, sometimes accompanied by tongue biting, foaming at the mouth, and involuntary urination (see bladder sphincter). Contrary to common beliefs, seizures do not destroy brain cells. However, recurring seizures lead to a scarring of the corresponding area in the brain.
A person who has a grand mal seizure might lose consciousness and fall to the ground (see also “falling conflict” causing vertigo). In this case, the Epileptoid Crisis of the motor conflict is coupled with a conflict that corresponds to the sensory, post-sensory, or pre-motor sensory cortex, for example with a separation conflict, that typically generates an “absence” (fainting) during the Epileptoid Crisis. This would explain why “absence seizures” are more common in children than adults. In a petit mal seizure, the zoning out only lasts a few seconds.
Recurring seizures are triggered by conflict relapses through setting on a track that was established when the motor conflict first took place. The “warning signs” preceding a seizure, called an epileptic aura, can become additional tracks, prompting further seizures. At that point, the original motor conflict might already be irrelevant.
Parkinson’s with tremors in one hand or both originates in a motor conflict associated with the hand(s). The tremors might also occur in the neck or in just one arm or leg, depending on the nature of the motor conflict. The typical muscle stiffness (rigidity) and slowing of movements (bradykinesia) are symptoms of a prolonged healing phase while the tremors occur during the Epileptoid Crisis. Permanent tremors, for example in the hands, are a sign of a continuous healing crisis due to constant motor conflicts of not being able to use the hands properly. In other words, the tremor itself is a track leading to a chronic condition. Conventional medicine considers Parkinson’s a “progressive neurodegenerative brain disorder” (a lack of dopamine is made responsible for the onset of the disease). Like with MS and ALS, the real reason why the condition advances is the negative prognosis and the fear of becoming completely disabled leading to additional motor conflicts and a worsening of the symptoms. Speech problems and voice changes are brought on by scare-fright conflicts.
Focal dystonia is a sustained muscle contraction (lasting Epileptoid Crisis) with repetitive movements of a specific muscle. In focal hand dystonia the finger or fingers – usually of one hand (handedness!) - curl into the palm or extend outward. The condition occurs most common among surgeons, dentists, and musicians, since people whose profession or hobbies require fine motor skills are more likely to experience a motor conflict associated with the finger(s) and hand(s) (compare with Dupuytren's contracture, a hand deformity related to the connective tissue). In sports such as tennis, baseball, or golf, the wrist spasms are commonly called yips. In cervical dystonia, also referred to as muscular torticollis or “wry neck”, the muscles around the neck contract intermittently, forcing the head to tip to one side with the chin thrust upwards. The underlying cause is a neck-related motor conflict. Generalized dystonia affecting most or all of the body presents as twisting of the limbs, specifically of the foot and leg or hand and arm, or of the torso (called Oppenheim’s disease). It is wrongly believed to be a “genetic disorder”. In people with Parkinson’s dystonia often arises from the effect of using the medication Levodopa (L-dopa).
Tetanus is characterized by muscle stiffness and body spasms. Tetanus is thought to be caused by nerve toxins, produced by the bacterium clostridium tetani that presumably enters the central nervous system through a wound. According to the medical literature, a “local tetanus”, in which patients have muscle contraction in the area of the injury, might be followed by a “generalized tetanus”. In GNM terms, the seizure-like muscle cramping takes place during the Epileptoid Crisis of a motor conflict that occurred during the fall which led to the injury. If anything, bacteria assist healing. Tetanus vaccinations might prevent “tetanus” but not the symptoms!
STROKE with motor paralysis
According to conventional medicine, the main causes of a stroke are:
In GNM, we differentiate between a sympathicotonic stroke (“white stroke”) and a vagotonic stroke (“red stroke”).
The white stroke occurs at the moment of the DHS. The impact of the motor conflict in the motor cortex generates sudden muscle weakness in one or more limbs, typically on one side of the body. Which side is affected is determined by a person’s handedness and whether the conflict is mother/child or partner-related. At this point, the weakness of the muscle(s) might be diagnosed as MS or ALS. However, an intense conflict leads quickly to muscle paralysis, possibly with paralysis of the facial muscles, including the tongue, affecting speech and swallowing (see Bell’s palsy). Now, the condition is called a “stroke”. Difficulties formulating words, termed Broca’s aphasia, involves the motor center for speech, known as the Broca’s area, located on the left side of the cerebral cortex in the brain relay that controls the laryngeal and vocal cord muscles. Hence, in people with Broca’s aphasia, the paralysis is always on the right side of the body. Numbness (sensory paralysis) in the face, arm and/or leg points to an additional separation conflict.
The muscle weakness or paralysis lasts throughout the conflict-active phase (cold hands, little appetite) and reaches into PCL-A. The Epileptoid Crisis, which is the period when the brain edema in the motor cortex is expelled, triggers uncontrolled jerking and contractions of the affected muscle(s) or an epileptic seizure. This is why it is sometimes difficult to tell strokes and seizures apart.
The red stroke takes place when a brain edema in close vicinity to the motor cortex presses onto the motor cortex, for example, an edema in the brain relay of the bronchi, larynx, or the myocardium. The “stroke” is initiated at the onset of the Epileptoid Crisis and lasts throughout the crisis from a few minutes (“transient ischemic attack”) to a few hours, depending on how long it takes to expel the edema. Impaired vision following a stroke occurs when a brain edema injures the optic nerve that runs through the cerebrum. After the Epileptoid Crisis, in PCL-B, the paralysis recedes and the motor ability slowly returns to normal. However, if the brain edema cannot be completely expelled, the paralysis (partly) remains since the synapses between the neurons don’t connect properly. This usually happens because of water retention due to an active abandonment and existence conflict (the SYNDROME) where water is also stored in the area of the brain that is healing at the time. Permanent paralysis can also be the result of repetitive scarification processes in the motor cortex due to continuous conflict relapses.